The Covid-19 spikes attache to angiotensin converting enzyme (ACE) receptors on the cell surfaces and gain entry into the cells where it migrates into the nucleus and its RNA attaches to our DNA, killing and altering the cells. A theory holds that ACE inhibitors (lisinopril, benzopril, and enalopril for example) and ace receptor blockers (losartan, valsartan, and olmesartan, for example) may inhibit entry of virus into our cells.
The Covid-19 spikes attache to angiotensin converting enzyme (ACE) receptors on the cell surfaces and gain entry into the cells where it migrates into the nucleus and its RNA attaches to our DNA, killing and altering the cells. A theory holds that ACE inhibitors (lisinopril, benzopril, and enalopril for example) and ace receptor blockers (losartan, valsartan, and olmesartan, for example) may inhibit entry of virus into our cells.
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